Upper Molar Dens in Dente - Case Report
Wanderley
F. COSTA
Manoel
D. SOUSA NETO
Jesus
D. PECORA
Trabalho publicado
no Brazialian Dental Journal
1(1):45-49, 1990
The
authors present a case report of dens in dente in an upper molar,
in which endodontic treatment was impossible due to its location.
Key
Words: dens in dente, dens invaginatus, therapy.
Introduction
Dens
in dente is an anomaly of development resulting from deepening or
invagination of the enamel organ into the dental papilla, which begins
at the crown and often extends to the root, before the calcification of
the dental tissues.
The
first observation of dens in dente dates back lo 1856 (De Smit and
Demaut, 1982). This anomaly is also known as dens invaginatus, dilated
odontome and gestant anomaly. According to Pécora et al. (1990),
the varied nomenclature occurs probably due to the lack of consensus in
relation to the cause of this alteration and the various names reflect
the different opinions as to its etiology.
Hovland
(1977) calculated the incidence of dens in dente to be 0.04 to 10.00%,
occurring in any tooth but with a greater frequency in the permanent upper
lateral incisors. Cases of bilateral occurrence have been reported (Swanson
and McCarthy, 1947; Grossman, 1976; Burton et al., 1980).
Dens
in dente is classified into 3 types, depending on the depth of invagination.
Type I, the invagination ends in a blind sac, limited to the dental crown.
Type II, the invagination extends to the amelocemental junction, also ending
in a blind sac. Type III, the invagination extends to the interior of the
root, providing an opening to the periodontium, sometimes presenting another
foramen in the apical region of the tooth (Oehlers, 1957).
Radiographically,
dens in dente shows a radiopaque invagination similar in density
to dental enamel (Goaz and While, 1987).
Maisto
(1973), Tagger (1977), Cole et al. (1978), Eldeeb (1984), Pécora
et al. (1987, 1990), and Vajrabhaya (1989) report cases of dens in dentetreated
with conventional endodontic methods. However, Fergunson et al. (1980),
Schindler and Walker (1983), and Pécora et al. (1987, 1990) describe
special endodontic techniques capable of inducing an apexification in Oehlers’
Type II dens in dente. Weine (1982) and Leonardo et al. (1982) report
the endodontic treatment of dens in dente as being a difficult operation,
preferring surgical treatment with retrograde filling.
Case
Report
A
26 year old, white, female patient was referred to our clinic with pain
in the upper left region. Clinical examination showed edema of the vestibular
cul-de-sac region, at the level of the second upper left molar, which did
not respond to thermal tests (heat and cold) but responded positively to
palpation and percussion.
An
x-ray showed the presence of Type III (Oehlers, 1957) dens in denteof the
second upper left molar with an apical radiolucent area (Figure
1).
Due
to the localization of the tooth and the impossibility of endodontic root
canal treatment, complemented by the retrograde filling of the apical opening,
the treatment of choice was extraction (Figures
2 and 3).
Discussion
The
presence of dens in dente is more common in the lateral upper incisors,
being rare in molars. Types I and II (Oehlers, 1957) dens in dente do
not present problems in endodontic treatment. In Type II, the invagination
is restricted to the interior of the root canal without reaching the apical
region of the canal, ending in a single foramen.
Pécora
et al. (1987, 1900), Maisto (1973) and Tagger (1977) present methods for
the endodontic treatment of Type II dens in dente.
Hata
and Toda (1987), Bolanos (1988) and Kulild (1989) recommend the endodontic
treatment of dens in dente in anterior upper teeth, even in Type
III (Oehiers, 1957) cases. In these cases, conventional endodontic treatment
in the area of invagination must be complemented by retrograde filling
of the foramen of the principal canal.
In
the case reported here, the first upper left molar presented an apical
radiolucent area and pulp necrosis, probably due to the fact that dens
in dente, Types II and III (Oehlers, 1957), permits the penetration
of irritants into the interior pulp tissue once the invagination establishes
this communication with the buccal cavity. Contamination can also occur
by communication with the pulp through the cul-de-sac of the invagination
causing tissue necrosis.
The
current literature does not present solutions for the treatment of Type
III dens in dente in molars which will only be possible when the
retrograde filling of the root canal is viable.
References
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496-499,1980
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GM, Taintor JF, James GA: Endodontic therapy of a dilated dens invaginatus.
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ME: Nonsurgical endodontic therapy of a dens invaginatus. J Endodont
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